| Weight | 329.487 g/mol |
|---|---|
| Formula | C24H27N |
| Hydrogen Acceptors | 1 |
| Hydrogen Donors | 1 |
| Aromatic Rings | 3 |
| Rotatable Bonds | 8 |
PRENYLAMINE (390-64-7)
Corontin · Difril · Segontin
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- The inhibition of the sarcoplasmic calcium pump by prenylamine, reserpine, chlorpromazine and imipramine (Naunyn-schmiedebergs Archives of Pharmacology, 1968)
- Inhibitory effects of verapamil, prenylamine and D 600 on Ca2+-dependent noradrenaline release from the sympathetic nerves of isolated rabbit hearts (Naunyn-schmiedebergs Archives of Pharmacology, 1979)
- Amelioration of adriamycin-induced cardiotoxicity in rabbits by prenylamine and vitamins A and E. (American Heart Journal, 1986)
- The binding of the calcium transport inhibitors reserpine, chlorpromazine and prenylamine to the lipids of the membranes of the sarcoplasmic reticulum (Naunyn-schmiedebergs Archives of Pharmacology, 1968)
- Studies on drug-induced lipidosis: VII. Effects of bis--diethylaminoethylether of hexestrol, chloroquine, homochlorocyclizine, prenylamine, and diazacholesterol on the lipid composition of rat liver and kidney (Lipids, 1976)
- Calcium and stimulus-secretion coupling in the neurohypophysis. II. Effects of lanthanum, a verapamil analogue (D600) and prenylamine on 45-calcium transport and vasopressin release in isolated rat neurohypophyses. (European Journal of Endocrinology, 1974)
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Predict GHS Hazards for Any Chemical in silico.
Including Acute Oral Tox, Skin Sensitization, Eye Irritation, Aquatic Tox, & more. - Corontin
- Difril
- Segontin
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SMILESCC(CC1=CC=CC=C1)NCCC(C2=CC=CC=C2)C3=CC=CC=C3
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InChIKeyIFFPICMESYHZPQ-UHFFFAOYSA-N
- Pubchem - PRENYLAMINE
- Wikipedia - prenylamine
Prenylamine (Segontin) is a calcium channel blocker of the amphetamine chemical class which was used as a vasodilator in the treatment of angina pectoris; it was introduced in the 1960s by German manufacturer Albert-Roussel pharma gmbh which was acquired by Hoechst AG in 1974 and which in turn became part of Sanofi Aventis in 2005. It was withdrawn from market worldwide in 1988 because it caused QT prolongation and Torsades de pointes which in turn caused sudden death. The cardiac side effects were not detected during clinical development, but only emerged after the drug was widely used.
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